Message from @lillian
Discord ID: 790920613144952892
no
don’t try to run away from the topic
that isn’t what u talked abt
I am not running away from the topic
u said that this topic had nothing to do w neurology. if ur studying someone’s brain, no matter who they are, it literally is neurology
Your claim was absurd so I questioned whether you know what neurology is or not
Communism is based
and then u went on to say that my previous discussion had nothing to do w neurology
just admit ur an idiot and leave
R u ignoring me
Wtf
omg no
You are the idiot here
depends but yes !!
They don't support trans
Cool
that isn’t what the discussion is abt tho
It actually is
...
Yes
That's why it has nothing to do with supporting trans
but that’s a different discussion
What did you mean when you said “neurology supports trans people”? @lillian
according to many different studies, the brain structure and behavior of trans women is similar to that of cis women and a lot closer to cis women rather than cis men. it supports the claim that trans women are women
Show me those studies
i sent like
12 studies last night
No you haven't
swipe up
...
these are 8 of them
yeah dude u need therapy
here are the rest
1st
Our data reveal a sex-atypical INAH3 volume and neuron number in transsexual male-to-female people to be in the female range, while the values of a female-to-male subject were in the male range. Differences in adult testosterone levels can only partly explain the observed differences in the INAH3 subdivision of transsexual people while estrogen levels do not seem to have an influence. In male-to-female subjects the number of neurons in the INAH3 does not seem to be related to sexual orientation, nor to the onset time of transsexuality, but rather to atypical early female-biased gender.
2nd
Conclusions: Although the mechanisms remain to be determined, there is strong support in the literature for a biologic basis of gender identity
Conclusions from 2 of the papers what you sent
3rd
Our results might theoretically also be explained by a lack of androgens in the transsexual group because all subjects, except for T4, had been orchiectomized. We, therefore, studied two nontranssexual men (S3 and S5) who had been orchiectomized because of prostate cancer 3 months and 2 yr before death, respectively, and found that the BSTc neuron number of S3 was close to the mean of the male group and that the BSTc number of neurons of S5 was even the highest observed (Fig. 1), indicating that orchiectomy did not cause any decrease in SOM neuron numbers. Not only were five of the transsexuals orchiectomized, they all used the antiandrogen cyproterone acetate (CPA)
4th
Results revealed that regional gray matter variation in MTF transsexuals is more similar to the pattern found in men than in women